Перевод контекст "dermatitis" c английский на русский от Reverso Context: However, I detect contact dermatitis on her knees. to TBT has resulted in irritation of the upper respiratory tract, severe dermatitis and eye irritation. syndrome, allergic contact dermatitis, angioedema, mastocytosis, lupus erythematosus. Allergic conjunctivitis — Allergic eye disease Classification and external Allergic contact dermatitis — A red, itchy, weepy reaction where the skin has come. 6 Usual but very effective Natural Remedies to eliminate Under Eye Dark Circles FOREVER - Eve's Special(Baking Face Under Eyes).
Is it possible for contact dermatitis to develop within that window? Тем не менее, я обнаружил контактный дерматит на ее коленях. На основании Contact dermatitus eyes allergy запроса эти примеры могут содержать разговорную лексику. Возможно ли, что контактный дерматит развился в течение этого отрезка времени? How do you solve a problem like dermatitis? Зарегистрируйтесь, чтобы увидеть больше примеров. Dermatitis — Inflammation of the skin, either due to direct contact with an irritating substance, or to an allergic reaction.
Drug Eyelid Dermatitis
Неонатальный себорейный дерматит - нередкое состояние, проявляющееся в виде твердых желтоватых чешуек на коже головы младенца. Как решить такую проблему, как дерматит? Экспорт словарей на сайты , сделанные на PHP,. Пометить текст и поделиться Искать во всех словарях Искать в переводах Искать в Интернете. Just the wound, which is not unlike rhus dermatitis. There are two types of dermatitis: Differential diagnoses to consider are carcinoid syndrome, allergic contact dermatitis , angioedema, mastocytosis, lupus erythematosus. Что такое контекстный словарь Скачать приложение Контакты Правовые вопросы. Перевод Словарь Проверка орфографии Спряжение Грамматика. Allergic inflammation — is an important pathophysiological feature of several disabilities or medical conditions including allergic asthma, atopic dermatitis, allergic rhinitis and several ocular allergic diseases. Occupational exposure of workers to TBT has resulted in irritation of the upper respiratory tract, severe dermatitis and eye irritation.
Смотреть что такое "allergic dermatitis" в других словарях: Неуверенность усиливает мой дерматит. К серьезным последствиям относятся возбуждение центральной нервной системы, судороги, нарушение дыхания, отек легких и дерматит. Occupational exposure of workers to TBT has resulted in irritation of the upper respiratory tract, severe dermatitis and eye irritation. На основании Вашего запроса эти примеры могут содержать грубую лексику. Возможно ли, contact dermatitus eyes allergy, что контактный дерматит развился в течение этого отрезка времени?
Aug 02, Author: The longer an individual has severe dermatitis, contact dermatitus eyes allergy, the longer, it is believed, that the dermatitis will take to resolve once the cause is identified. See 5 Body Modifications and Piercing: Dermatologic Risks and Adverse Reactionsa Critical Images slideshow, to help recognize various body modifications and the related potential complications.
Acute allergic contact dermatitis is characterized by pruritic papules and vesicles on an erythematous base. Lichenified pruritic plaques may indicate a chronic form of the condition. Individuals with allergic contact dermatitis typically develop the condition within a few days of exposure, in areas that were exposed directly to the allergen. Certain allergens eg, neomycinhowever, penetrate intact skin poorly; in such cases, the onset of dermatitis may be delayed for up to a week following exposure.
Individuals may develop widespread dermatitis from topical medications applied to leg ulcers or from cross-reacting systemic medications administered intravenously. Contact dermatitus eyes allergy metal contact allergy may result in mucositis that mimics lichen planus, which has an association with contact dermatitus eyes allergy squamous cell carcinoma.
See Clinical Presentation for more detail. To exclude tinea; these tests are often indicated for dermatitis of contact dermatitus eyes allergy hands and feet.
Repeat open application test ROAT: To determine whether a metallic object contains enough nickel to provoke allergic dermatitis. May help to exclude other disorders, particularly tinea, psoriasis, and cutaneous lymphoma. The definitive treatment for allergic contact dermatitis is the identification and removal of any potential causal agents; otherwise, the patient is at increased risk for chronic or recurrent dermatitis.
Treatments also include the following:. Topical corticosteroids are the mainstay of treatment, although acute, severe allergic contact dermatitis, such as from poison ivy, often needs to be treated with a 2-week course of systemic corticosteroids. Approved for atopic dermatitis, but they are also prescribed for cases of allergic contact dermatitis when they offer safety advantages over topical corticosteroids.
Administered to individuals with chronic allergic contact dermatitis that is not controlled well by topical corticosteroids; these patients may benefit from treatment with a combination of psoralen a photosensitizer and ultraviolet-A PUVA, contact dermatitus eyes allergy. Chronic immunosuppressive agents are, in rare instances, used to treat recalcitrant cases of severe, chronic, widespread allergic contact dermatitis or severe hand dermatitis that prevents a patient allergy rash red face working or performing daily activities.
Occasionally, contact dermatitus eyes allergy, an individual who is highly allergic to nickel and has severe vesicular hand dermatitis will benefit from treatment with disulfiram Antabuse ; the drug has a chelating effect.
See Treatment and Medication for more detail. Allergic contact dermatitis ACD is a delayed type of induced sensitivity allergy resulting from cutaneous contact with a specific allergen to which the patient has developed a specific sensitivity.
This allergic reaction contact dermatitus eyes allergy inflammation of food allergy rashes skin manifested by varying degrees of erythema, edema, and vesiculation. The term contact dermatitis sometimes is used incorrectly as a synonym for allergic contact dermatitis.
Contact dermatitis is inflammation of the skin induced by chemicals that directly damage the skin see Irritant Contact Dermatitis and by specific sensitivity in the case of allergic contact dermatitis. Jadassohn first described allergic contact dermatitis in He developed the patch test to identify the chemicals to which the patient was allergic.
Sulzberger popularized patch testing in the United States in the s. The Finn chamber method for patch testing was designed in the s; these chambers consist of small metal cups, typically attached to strips of tape, filled with allergens dispersed in either petrolatum or water.
The importance of specific substances as causes of allergic contact dermatitis varies with the prevalence of that substance in the environment. Mercury compounds once were significant causes of allergic contact dermatitis but rarely are used as topical medications and, currently, are uncommon as a cause of allergic contact dermatitis.
Ethylenediamine, which was present in the original Mycolog cream, declined as a primary cause of allergic contact dermatitis once Mycolog cream was reformulated to no longer contain this allergen. A detailed history, both before and after patch testing, is crucial in evaluating individuals with allergic contact dermatitis.
Before patch testing, the history identifies potential causes of allergic contact dermatitis and the materials to which individuals are exposed that should be included in patch testing. After patch testing, the history determines the clinical significance of the findings. Topical corticosteroids are the mainstay of treatment, while a variety of symptomatic treatments can provide short-term relief of pruritus.
However, the definitive treatment of allergic contact dermatitis is the identification and removal of any potential causal agents; otherwise, the patient is at increased risk for chronic or recurrent dermatitis. Approximately chemicals are well documented as specific causes of allergic contact dermatitis.
Compounds must be less than d for efficient penetration through the stratum corneum barrier, which is the water-impermeable outer layer of the contact dermatitus eyes allergy. Small organic molecules that are chemically reactive chemical sensitizers bind with self-proteins to generate immunogenic neoantigens through a contact dermatitus eyes allergy termed haptenization.
Although haptens can penetrate through intact skin, patients with certain disease states that impair barrier function eg, leg ulcers, perianal dermatitis have an increased risk of sensitization to topically applied medications and their vehicle components.
Allergy tongue ring patients with atopic dermatitis or allergic contact dermatitis to nickel harbor a defective form of the filaggrin gene. In its absence, the barrier is defective. Prehaptens are chemicals that are not activated by host proteins, but instead food allergies and skin rash chemical transformation by oxidative derivatization by ambient or air oxidation to form hydroperoxide, contact dermatitus eyes allergy.
Examples british medical journal cherkasky peanut allergy certain fragrance materials and dyes used in hair coloring, contact dermatitus eyes allergy, such as para-phenylenediamine, contact dermatitus eyes allergy. Contact dermatitus eyes allergy activate Toll-like receptors TLRs and activate innate immunity. The importance of hapten-mediated activation of innate immunity is highlighted by the clinical observation that the irritancy of chemicals ie, the ability of these chemicals to cause grossly visible skin inflammation upon primary exposure correlates with their ability to act as contact sensitizers and to induce acute contact dermatitis.
As a result, skin-resident dendritic cells DCs become activated. There are several populations of DCs. Langerhans cells are the only DC subtype in the epidermis.
In the final step, hapten-induced inflammation recruits activated effector T cells back to the initial site of antigen encounter in the skin. Keratinocytes are crucial for contact dermatitus eyes allergy development of allergic contact dermatitis. They constitute the vast majority of cells in the epidermis and form the anatomic barrier of the skin.
Keratinocytes are also a source of IL, an immunosuppressive cytokine that limits the extent of contact hypersensitivity. The initial sensitization typically takes days from initial exposure to a strong contact allergen such as poison ivy. Some individuals develop specific sensitivity to allergens following years of chronic low-grade exposure; for example, sensitivity to chromate in cement can eventually develop in individuals with chronic irritant contact dermatitis resulting from the alkaline nature of cement.
Once an individual is sensitized to a contact dermatitus eyes allergy, allergic contact dermatitis develops within hours to several days of exposure. Approximately 25 chemicals appear to be responsible for as many as one half of all cases of allergic contact dermatitis.
These include nickel, preservatives, dyes, and fragrances. Poison ivy Toxicodendron radicans is the classic example of acute allergic contact dermatitis in North America, contact dermatitus eyes allergy.
Allergic contact dermatitis from poison ivy is characterized by linear streaks of acute dermatitis that develop where plant parts have been in direct contact with the skin. Nickel is the leading cause of allergic contact dermatitis in the world. The incidence of nickel allergic contact dermatitis in North America is increasing; in contrast, new regulations in Europe have resulted in a decreasing prevalence of nickel allergy in young and middle-aged women, contact dermatitus eyes allergy.
Allergic contact dermatitis to nickel typically is manifested by dermatitis at the sites where earrings or necklaces see the image below containing nickel are worn or where metal objects including the keypads of some cell phones [ 4 ] containing nickel are in contact with the skin. Nickel may be considered a possible occupational allergen. Workers in whom nickel may be an occupational allergen primarily include hairdressers, retail clerks, caterers, domestic cleaners, and metalworkers.
Individuals allergic to nickel occasionally may develop vesicles on the sides of the fingers dyshidrotic hand eczema or pompholyx from nickel in the diet. Allergy to 1 or more chemicals in rubber gloves is suggested in any individual with chronic hand dermatitis who wears them, unless patch testing demonstrates otherwise.
Allergic contact dermatitis to chemicals in rubber gloves typically occurs maximally on the dorsal aspects of the hand. Usually, a cutoff of dermatitis occurs on the forearms where skin is no longer in contact with the gloves. Individuals allergic to chemicals in rubber gloves may develop dermatitis from other exposures to the chemicals eg, under elastic waistbands.
Severe local reactions from PPD may occur in black henna tattoos in adults and children. Epidemiologic data indicate that the median prevalence of positive patch test reactions to Contact dermatitus eyes allergy among dermatitis patients is 4. Individuals allergic to dyes and permanent press and wash-and-wear chemicals added to textiles typically develop dermatitis on the trunk, which occurs maximally on the lateral contact dermatitus eyes allergy of the trunk but spares the vault of the axillae.
Primary lesions may be small follicular papules or may be extensive plaques. Individuals in whom this allergic contact dermatitis is suspected should be tested with a series of textile chemicals, particularly if routine patch contact dermatitus eyes allergy reveals no allergy to formaldehyde. New clothing is most likely to provoke allergic contact dermatitis, since most allergens decrease in concentration in clothing following repeated washings. Preservative chemicals added to cosmetics, moisturizers, contact dermatitus eyes allergy, and topical medications are major causes of allergic contact dermatitis see the image below.
The risk of allergic contact dermatitis appears to be highest to quaternium, followed by allergic contact dermatitis to isothiazolinones. Methylisothiazolinone is used as an individual preservative and may be a significant allergen. Although parabens contact dermatitus eyes allergy among the most widely used preservatives, they are not a frequent cause of allergic contact dermatitis. Schnuch et al estimated that preservatives found in leave-on topical products varied over 2 orders of magnitude in relative sensitization risk.
Formaldehyde is a major cause of allergic contact dermatitis see the image below. Certain preservative chemicals widely used in shampoos, lotions, other moisturizers, and cosmetics are termed formaldehyde releasers ie, quaternium [Dowicil ], imidazolidinyl urea [Germall ], and isothiazolinones [ 9 ]. They are, in themselves, allergenic or may produce cross-sensitization to formaldehyde. Individuals may develop allergy to fragrances. Fragrances are found not only in perfumes, colognes, aftershaves, deodorants, and soaps, but also in numerous other products, often as a mask to camouflage an unpleasant odor.
Unscented products may contain fragrance chemicals used as a component of the product and not labeled as fragrance. Individuals allergic to fragrances should use fragrance-free products. Unfortunately, the exact chemicals responsible for a fragrance in a product are not labeled. Four thousand different fragrance molecules are available to formulate perfumes. The fragrance industry is not required to release the names of ingredients used to compose a fragrance in the United States, even when individuals develop allergic contact dermatitis to fragrances found in topical medications.
Deodorants may be the most common cause of allergic contact dermatitis to fragrances because they are applied to occlude skin that is often abraded by shaving in women. Massage and physical therapists and geriatric nurses are at higher risk of contact dermatitus eyes allergy allergic contact dermatitis to fragrances. In the last decade, it has become clear that some individuals with chronic dermatitis develop allergy to topical corticosteroids.
Most affected individuals can be treated with some topical corticosteroids, but an individual can be allergic to all topical and systemic corticosteroids. Budesonide and tixocortol pivalate are useful patch test corticosteroids for identifying individuals allergic to topical corticosteroids.
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